2015). These research, taken with each other, recommend that, While a direct activation of GPR55 or PPAR‐α happens, PEA can create its anti‐inflammatory motion inside the gut also through oblique activation of CB1 and CB2 receptors, possibly on account of the power of the compound to potentiate the motion of endocannabinoids at these receptors
Within this evaluate, we focus on the molecular targets of PEA, its pharmacological activity in neurological Diseases, suffering notion and inflammatory illnesses, together with the present evidence over the performance of micronized and extremely‐micronized formulations of PEA when offered by itself or in combination Together with the antioxidan
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